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The purpose of this study was to further illustrate the relationship between baroreceptor reflex sensitivity (BRS) and hypertensive end-organ damage (EOD) and to test the hypothesis that impairment of BRS aggravates EOD in hypertension. ... The Medscape Journal ... Allergy & Clinical Immunology...
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www.medscape.com/medline/abstract/10484442
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The purpose of this study was to examine the effect of inhibition of Ras-GTPase mediated signalling on the development of hypertension and end-organ damage in spontaneously hypertensive rats chronically treated with nitric oxide synthesis inhibitor L-NAME (SHR-L-NAME). ... The Medscape Journal ... Allergy & Clinical Immunology...
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www.medscape.com/medline/abstract/16027001
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Hypertension - Wikipedia, the free encyclopedia
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Hypertension is a chronic medical condition in which the blood pressure is elevated. It is also referred to as high blood pressure or shortened to HT , HTN or HPN . The word "hypertension", b...
en.wikipedia.org/wiki/Hypertension
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Top; Abstract; Introduction; Hypertensive Mechanisms in dTGR; Vascular and End-Organ Damage; dTGR as a Model... dTGR as a Model... References; ... Hypertension injures blood vessels and thereby causes end-organ damage. The mechanisms are complicated and, although studied for decades in experimental animal models,1 are...
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hyper.ahajournals.org/cgi/content/full/33/1/212
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Conclusions— These results demonstrate a crucial role for p38 MAPK in hypertensive cardiac hypertrophy and end-organ damage. Interrupting its function with a specific p38 MAPK inhibitor halts clinical deterioration.
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circ.ahajournals.org/cgi/content/abstract/104/11/1292
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Long-term treatment with a selective p38 MAPK inhibitor, SB239063, provided a remarkable reduction in morbidity, mortality, and hypertensive end-organ damage and dysfunction.
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circ.ahajournals.org/cgi/content/full/104/11/1292
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hypertensive emergency: associated evidence of end-organ damage: hypertensive encephalopathy; stroke (cerebral infarction or intracerebral or subarachnoid haemorrhage); acute pulmonary oedema, congestive heart failure, left ventricular failure or aortic dissection;
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www.eboncall.org/CATs/2352.htm
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hypertensive emergency: associated evidence of end-organ damage: hypertensive encephalopathy; stroke (cerebral infarction or intracerebral or subarachnoid haemorrhage; acute pulmonary oedema, congestive heart failure, left ventricular failure or aortic dissection;
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www.eboncall.org/CATs/2353.htm
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Hypertensive end-organ damage and premature mortality are p38 mitogen-activated protein kinase-dependent in a rat model of cardiac hypertrophy and dysfunction. Paper-9062143. Little or no difference in blood pressure was noted in the treatment or vehicle groups.
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www.ihop-net.org/UniPub/iHOP/pm/9062143.html
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