In 31 segments of angiographically smooth arteries in group I, the CPT produced vasodilation from a control mean diameter of 2 ... G. Heper; Effects of Afterload Increase on Systolic and Diastolic Functions of the Myocardium After Myocardial Infarction; Angiology, March 1, 2004; 55(2): 159 - 167. [Abstract] [PDF];
circ.ahajournals.org/cgi/content/abstract/77/1/43
In stenotic arteries, as the artery constricts, the pressure (or afterload) opposing smooth muscle shortening decreases, leading to exaggerated constriction ...
linkinghub.elsevier.com/retrieve/pii/105017389290048W
Elevation of afterload with thoracic aortic constriction led to a ... was passed behind the aorta between the brachiocephalic and left carotid arteries. ...
linkinghub.elsevier.com/retrieve/pii/S0005276098001702
Afterload is the resistance the blood encounters when ejected from the heart—remember how arteries constrict like nozzles?] ... The second thing you need to do after increasing your Preload, is reduce your Afterload. This means reducing the resistance the blood encounters. The best Afterload-reducing agent I know of...
www.dfwcfids.org/medical/cheney/heart04.part2b.htm
In contrast to the systemic arteries, pulmonary vessels constrict with hypoxia (Euler-Liljestrand reflex) and relax in the presence of hyperoxia. Furthermore, changes in cardiac output (CO), airway pressure, and gravity affect ... In contrast to LV performance, RV function is relatively sensitive to increases in afterload.
www.anesthesia-analgesia.org/cgi/content/full/96/6/1603
During dynamic exercise, normal coronary arteries dilate, whereas stenotic arteries constrict. ... Pharmacological treatment in patients with stable angina pectoris may improve myocardial ischemia by reducing pre- and afterload, myocardial contractility, oxygen consumption, and vasomotor tone.
www.unboundmedicine.com/medline/ebm/record/10860181/abs... www.unboundmedicine.com/medline/ebm/record/10860181/abstract/Impact_of_exercise_induced_coronary_vasomotion_on_anti_ischemic_therapy_
The decrease in output forces the body to compensate by activating angiotensin-converting enzyme (ACE) to excessive levels, forming angiotensin II, which causes the veins and arteries to constrict. Angiotensin II also releases aldosterone, resulting in sodium and water retention.
www.cavalierhealth.org/mitral_valve_disease.htm
NO-mediated dilation of feed arteries permits increased microvascular flow without reduction in muscle perfusion pressure. ... At the same time, aortic stiffening increases systolic pressure and cardiac afterload, further stressing the relationship between cardiac systolic performance and myocardial perfusion.
www.fasebj.org/cgi/content/full/14/12/1685
Afterload is the resistance the blood encounters when ejected from the heart – remember how arteries constrict like nozzles?] [The diagram could be seen as plotting the amount of blood waiting in the ventricle to go to the body (horizontal axis) against the amount of blood that is actually ejected from the...
www.immunesupport.com/library/showarticle.cfm?id=6683&T... www.immunesupport.com/library/showarticle.cfm?id=6683&T=CFIDS_FM
More modest effects on the arterial circulation decrease systolic wall stress (afterload), contributing to further reductions in MVO2. ... NTG dilates normal and atherosclerotic epicardial coronary arteries as well as smaller arteries that constrict with certain stressors (e.g., cold, mental or physical exercise).
www.acc.org/qualityandscience/clinical/guidelines/unsta... www.acc.org/qualityandscience/clinical/guidelines/unstable/incorporated/III_hospital.htm